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Nrf2-Mediated HO-1 Induction Coupled with the ERK Signaling Pathway Contributes to Indirect Antioxidant Capacity of Caffeic Acid Phenethyl Ester in HepG2 Cells

机译：Nrf2介导的HO-1诱导结合ERK信号通路对HepG2细胞中咖啡酸苯乙酯间接抗氧化能力的贡献

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Jin-Kyoung Kim; Hae-Dong Jang;
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1. Nrf2-Mediated HO-1 Induction Coupled with the ERK Signaling Pathway Contributes to Indirect Antioxidant Capacity of Caffeic Acid Phenethyl Ester in HepG2 Cells [J] . Hae-Dong Jang, Jin-Kyoung Kim International Journal of Molecular Sciences . 2014,第7期

机译：Nrf2介导的HO-1诱导与ERK信号通路耦合，促进HepG2细胞中咖啡酸苯乙基酯的间接抗氧化能力。
2. Caffeic Acid Phenethyl Ester (CAPE) Protects PC12 Cells Against Cisplatin-Induced Neurotoxicity by Activating the AMPK/SIRT1, MAPK/Erk, and PI3k/Akt Signaling Pathways [J] . Ferreira Rafaela Scalco, Guinaim dos Santos Neife Aparecida, Bernardes Carolina P., Neurotoxicity research . 2019,第1期

机译：通过激活AMPK / SIRT1，MAPK / ERK和PI3K / AKT信号通路，通过激活AMPK / SIRT1，MAPK / ERK和信号传导途径来保护PC12细胞免受顺铂诱导的神经毒性的影响
3. Antioxidant mechanism of polyphenol-rich Nymphaea nouchali leaf extract protecting DNA damage and attenuating oxidative stress-induced cell death via Nrf2-mediated heme-oxygenase-1 induction coupled with ERK/p38 signaling pathway [J] . Bajpai Vivek K., Alam Md Badrul, Ju Mi-Kyoung, Biomedicine & pharmacotherapy =: Biomedecine & pharmacotherapie . 2018,第期

机译：富含多酚富含Nophaea Nouchali叶提取物的抗氧化机制保护DNA损伤并通过NRF2介导的血红素氧酶-1诱导诱导氧化胁迫诱导的细胞死亡，与ERK / P38信号通路偶联
4. Induction of superoxide by 12-O-tetradecanoylphorbol-13-acetate and thapsigargin, a non-phorbol ester-type tumor promoter, in peritoneal macrophages elicited from SENCAR and B6C3F1 mice: The significance of protein kinase C vs. arachidonic acid-mediated signal transduction pathways [D] . Yoon, Hyunah Lee 1992

机译：由SENCAR和B6C3F1小鼠引起的腹膜巨噬细胞中12-O-十四烷酰佛波醇13-乙酸盐和thapsigargin，一种非佛波醇酯型肿瘤启动子的超氧化物诱导作用：蛋白激酶C与花生四烯酸介导的信号转导的意义途径
5. Nrf2-Mediated HO-1 Induction Coupled with the ERK Signaling Pathway Contributes to Indirect Antioxidant Capacity of Caffeic Acid Phenethyl Ester in HepG2 Cells [O] . Jin-Kyoung Kim, Hae-Dong Jang 2014

机译：Nrf2介导的HO-1诱导与ERK信号通路耦合促进HepG2细胞中咖啡酸苯乙基酯的间接抗氧化能力。
6. Caffeic Acid Phenethyl Ester Causes p21Cip1 Induction, Akt Signaling Reduction, and Growth Inhibition in PC-3 Human Prostate Cancer Cells [O] . Lin, Hui-Ping, Jiang, Shih Sheng, Chuu, Chih-Pin 2012

机译：咖啡酸苯乙基酯在PC-3人前列腺癌细胞中引起p21Cip1诱导，Akt信号传导减少和生长抑制
7. Bifunctional Alkylating Agent-Induced p53 and Nonclassical Nuclear Factor kB Responses and Cell Death are Altered by Caffeic Acid Phenethyl Ester: A Potential Role for Antioxidant/Electrophilic Response-Element Signaling [R] . Minsavage, G. D. , Dillman, J. F. 2007

机译：双功能烷化剂诱导的p53和非经典核因子KB反应和细胞死亡由咖啡酸苯乙酯改变：抗氧化/亲电反应元素信号的潜在作用

Nrf2-Mediated HO-1 Induction Coupled with the ERK Signaling Pathway Contributes to Indirect Antioxidant Capacity of Caffeic Acid Phenethyl Ester in HepG2 Cells

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